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Cagrilintide

Weight loss, appetite reduction, glycemic control. Categorized as a Metabolic peptide.

Also known as: NN9838, AM833

Updated: 2026-04-03
3 cited studies

What Is Cagrilintide?

Cagrilintide (NN9838) is classified as a metabolic peptide. Long-acting amylin analog that activates amylin receptors in the area postrema and hypothalamus to reduce appetite, slow gastric emptying, and suppress post-meal glucagon secretion. Works on a distinct pathway from GLP-1 agonists, making it ideal for combination therapy.

It is extensively evaluated in laboratory and clinical settings for its potential to drive weight loss, appetite reduction, glycemic control. Researchers target Cagrilintide for its ability to interact with specific cellular and molecular pathways, making it a compound of significant interest across multiple therapeutic domains.

Generally well-tolerated in Phase 2 trials. Main side effects are GI-related (nausea, vomiting). Novo Nordisk is developing CagriSema (cagrilintide + semaglutide) as next-gen obesity treatment. Research-only peptide.

How Does Cagrilintide Work?

Long-acting amylin analog that activates amylin receptors in the area postrema and hypothalamus to reduce appetite, slow gastric emptying, and suppress post-meal glucagon secretion. Works on a distinct pathway from GLP-1 agonists, making it ideal for combination therapy.

At the molecular level, Cagrilintide operates through pathways characteristic of the Metabolic class. By interacting with target receptors and downstream signaling cascades, the compound initiates biological responses associated with weight loss, appetite reduction, glycemic control.

Expected Research Timeline

Weeks 2–4

Measurable weight loss (1-2 lbs/week); improved meal portion control

Months 2–3

10-11% body weight loss at clinical doses; improved glycemic markers

Long-Term

Clinical trials show sustained weight loss through 26+ weeks; CagriSema in Phase 3 trials

What Does the Research Say?

The following are key findings from peer-reviewed studies on Cagrilintide, indexed on PubMed and equivalent databases:

Safety & Side Effects

Generally well-tolerated in Phase 2 trials. Main side effects are GI-related (nausea, vomiting). Novo Nordisk is developing CagriSema (cagrilintide + semaglutide) as next-gen obesity treatment. Research-only peptide.

Side EffectIncidenceSeverity
Nausea~25% of usersmoderate
Diarrhea~10% of usersmild
Vomiting~8% of usersmoderate
Injection site reaction~5% of usersmild
Constipation~8% of usersmild

FDA Status: Not Approved for Human Therapeutic Use

Cagrilintide is not currently FDA-approved for human use. It is available for research purposes only. Always consult a licensed healthcare provider.

How Is Cagrilintide Used?

Route

SubQ

Dose Range

5004500 mcg

Frequency

Weekly

Cycle

12–24 wk

Timing: Any time, consistent day

Notes: Clinical trial doses: 0.3-4.5mg weekly with dose escalation over 4-8 weeks. Start low (0.3-0.6mg) and increase every 4 weeks to minimize GI side effects.

All dosing information reflects parameters reported in published research literature and is not intended as clinical guidance. Usage of any peptide should be supervised by a qualified healthcare professional.

Cagrilintide vs. Related Compounds

CompoundPrimary Use
Cagrilintide(this page)Weight loss, appetite reduction, glycemic control
MOTS-cEnergy, metabolism, fat loss, longevity
SemaglutideFat loss, appetite control, cardiovascular benefits
TirzepatideWeight loss, body recomposition, glycemic control

Where to Source Cagrilintide for Research

Purchasing ultra-high purity, laboratory-grade peptides is critical for verifiable research. We only recommend vendors providing independent, third-party HPLC Certificates of Analysis (COA).

View COA-Verified Cagrilintide
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Disclosure: PeptiDex may earn a commission from purchases. This does not affect our recommendations. We exclusively feature vendors that pass our strict quality verification protocols.

Frequently Asked Questions

What is Cagrilintide?

Cagrilintide is a metabolic peptide. Long-acting amylin analog that activates amylin receptors in the area postrema and hypothalamus to reduce appetite, slow gastric emptying, and suppress post-meal glucagon secretion. Works on a distinct pathway from GLP-1 agonists, making it ideal for combination therapy.

What are the primary research benefits of Cagrilintide?

Published research identifies primary mechanisms targeting: Weight loss, appetite reduction, glycemic control. These findings come from 3+ peer-reviewed studies indexed in our database.

What is the half-life of Cagrilintide?

In published pharmacokinetic data, Cagrilintide demonstrates a half-life of approximately 168 hours.

Is Cagrilintide FDA approved?

Cagrilintide is not currently FDA-approved for human therapeutic use. It is classified as a research compound and is studied under investigational protocols. Always consult a healthcare provider.

What are common side effects of Cagrilintide?

Reported side effects in published literature include Nausea (~25% of users), Diarrhea (~10% of users), Vomiting (~8% of users), Injection site reaction (~5% of users), Constipation (~8% of users). Most are classified as moderate in severity.

How is Cagrilintide administered?

In research settings, Cagrilintide is typically administered via SubQ. Clinical trial doses: 0.3-4.5mg weekly with dose escalation over 4-8 weeks. Start low (0.3-0.6mg) and increase every 4 weeks to minimize GI side effects.

Sources

  1. Cagrilintide Phase 2 trial for weight management. View on PubMed
  2. CagriSema (cagrilintide + semaglutide) Phase 2 results. View on PubMed
  3. Amylin analogs in obesity management: mechanism review. View on PubMed
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Medical Disclaimer

The information provided in this profile is for educational and research purposes only. Cagrilintide is not FDA-approved for human therapeutic use. Nothing on this page should be interpreted as medical advice. Always consult a licensed healthcare professional before interacting with any peptide compound. Read our full disclaimer.

Last updated: 2026-04-03 · About PeptideX · Editorial Standards